Congenital Heart Disease Quotes

Congenital disease can warp the heart with great variety. Valves can be sealed tight, missing parts—or absent altogether. Major vessels can be misplaced, narrowed, or blocked completely. A chamber can be too small or missing, a wall too thick or thin. The heart’s electrical system—its nerves—may go haywire. The muscle can be weak. Holes may occur almost anywhere, in almost any size. Studying heart pathology, one is reminded that the genetic symphony that produces a normal baby is indeed a wondrous and delicate one.

Surviving your child means moving ever forward away from the time when the person you loved more than you thought you were capable of loving was real and living in this world, and watching that vivid light fade to a darkness that leaves you permanently cold.

Meantime, I underwent another echocardiogram and a CT test with dye. Turns out, my condition had zero to do with diet or behavior. I was born with a congenital heart defect known as a bicuspid aortic valve, the valve that transports blood flow from the heart. It’s an inherited form of heart disease in which two of the leaflets of the valve fuse together in the womb. It is the most common cause of heart disease present at birth and affects between 1 and 2 percent of adults. The walls of the aorta are typically strong enough to tolerate the stress of blood flow from the heart. Aneurysms—which develop in about half of all patients with bicuspid valves—occur when the walls weaken. As the weakened wall deteriorates, it leaves behind damaged tissue that grows in size, heightening the risk of a serious tear. The aortic valve is supposed to be three centimeters. Mine? The size of a can of Coca-Cola. So that’s why time was of the essence.

leading to higher-than-normal levels of methemoglobin in the blood—a form of hemoglobin—that overwhelms the normal hemoglobin, which reduces oxygen capacity. Less oxygen in the blood makes it a chocolate-brown color instead of red, causing the skin to appear blue. Doctors can easily diagnose congenital methemoglobinemia because the color of the blood provides the unique clue. The mutation is hereditary and carried in a recessive gene, whereas acquired methemoglobinemia is life-threatening and derives from heart disease, airway obstruction, or taking too much of certain drugs.

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