Tissue Main Quotes

Saw a film on cancer yesterday, shown by the English delegation. No doubt about it. I'm right. "Migratory cancer cells" are amoebic formations. They are produced from disintegrating tissue and thus demonstrate the law of tension and charge in its purest form - as does the orgastic convulsion. Now money is a must - cancer the main issue - in every respect, even political. It was a staggering experience. My intuition is good. I depend on it. Was absolutely driven to buy a microscope. The sight of the cancer cells was exactly as I had previously imagined it, had almost physically felt it would be. Cancer is an autoinfection of the body, of an organ. And researchers have no idea of what, hor, or where!!

La plupart des gens que nous croisons sont quelconques, on n'en retient rien, sinon le regret du temps et de l'énergie perdus. Dans la constellation d'étoiles ternes qui traversent nos vies brille pourtant, à de rares et précieuses occasions, un soleil à portée de main. Parfois un parent, souvent un ami ou encore un professeur. Les plus chanceux en croiseront deux ou trois qui marqueront durablement leur personnalité, imprégneront un peu de magnificence dans le tissu de leur psyché.

But this metaphysics, like this science, has enfolded its deeper life in a rich tissue of symbols, forgetting something that, while science needs symbols for its analytical development, the main object of metaphysics is to do away with symbols.

When authors write, who do they describe: themselves, characters or the readers? But, characters cannot be the characters, because in every book the main character is writer slash author. Because they went through it before they wrote it." She finished wiping her mouth with napkin a d dropped her tissue beside her plate.

He handed me something done up in paper. 'Your mask,' he said. 'Don't put it on until we get past the city-limits.' It was a frightening-looking thing when I did so. It was not a mask but a hood for the entire head, canvas and cardboard, chalk-white to simulate a skull, with deep black hollows for the eyes and grinning teeth for the mouth. The private highway, as we neared the house, was lined on both sides with parked cars. I counted fifteen of them as we bashed by; and there must have been as many more ahead, in the other direction. We drew up and he and I got out. I glanced in cautiously over my shoulder at the driver as we went by, to see if I could see his face, but he too had donned one of the death-masks. 'Never do that,' the Messenger warned me in a low voice. 'Never try to penetrate any other member's disguise.' The house was as silent and lifeless as the last time - on the outside. Within it was a horrid, crawling charnel-house alive with skull-headed figures, their bodies encased in business-suits, tuxedos, and evening dresses. The lights were all dyed a ghastly green or ghostly blue, by means of colored tissue-paper sheathed around them. A group of masked musicians kept playing the Funeral March over and over, with brief pauses in between. A coffin stood in the center of the main living-room. I was drenched with sweat under my own mask and sick almost to death, even this early in the game. At last the Book-keeper, unmasked, appeared in their midst. Behind him came the Messenger. The dead-head guests all applauded enthusiastically and gathered around them in a ring. Those in other rooms came in. The musicians stopped the Death Match. The Book-keeper bowed, smiled graciously. 'Good evening, fellow corpses,' was his chill greeting. 'We are gathered together to witness the induction of our newest member.' There was an electric tension. 'Brother Bud!' His voice rang out like a clarion in the silence. 'Step forward.' ("Graves For Living")

Yatima found verself gazing at a red-tinged cluster of pulsing organic parts, a translucent confusion of fluids and tissue. Sections divided, dissolved, reorganised. It looked like a flesher embryo – though not quite a realist portrait. The imaging technique kept changing, revealing different structures: Yatima saw hints of delicate limbs and organs caught in slices of transmitted dark; a stark silhouette of bones in an X-ray flash; the finely branched network of the nervous system bursting into view as a filigreed shadow, shrinking from myelin to lipids to a scatter of vesicled neurotransmitters against a radio-frequency MRI chirp. There were two bodies now. Twins? One was larger, though – sometimes much larger. The two kept changing places, twisting around each other, shrinking or growing in stroboscopic leaps while the wavelengths of the image stuttered across the spectrum. One flesher child was turning into a creature of glass, nerves and blood vessels vitrifying into optical fibres. A sudden, startling white-light image showed living, breathing Siamese twins, impossibly transected to expose raw pink and grey muscles working side by side with shape-memory alloys and piezoelectric actuators, flesher and gleisner anatomies interpenetrating. The scene spun and morphed into a lone robot child in a flesher's womb; spun again to show a luminous map of a citizen's mind embedded in the same woman's brain; zoomed out to place her, curled, in a cocoon of optical and electronic cables. Then a swarm of nanomachines burst through her skin, and everything scattered into a cloud of grey dust. Two flesher children walked side by side, hand in hand. Or father and son, gleisner and flesher, citizen and gleisner... Yatima gave up trying to pin them down, and let the impressions flow through ver. The figures strode calmly along a city's main street, while towers rose and crumbled around them, jungle and desert advanced and retreated. The artwork, unbidden, sent Yatima's viewpoint wheeling around the figures. Ve saw them exchanging glances, touches, kisses – and blows, awkwardly, their right arms fused at the wrists. Making peace and melting together. The smaller lifting the larger on to vis shoulders – then the passenger's height flowing down to the bearer like an hourglass's sand.

From the start, one of the main factors in the discussion of heart disease has been cholesterol, the yellow, waxy substance that is a necessary part of all body tissues. It is a vital component of every cell membrane, controlling what goes in and out of the cell.

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The main areas for estrogen breakdown are the liver and gastrointestinal tract. Diets high in refined sugar and low in fiber feed the unfriendly bacteria in the intestines, causing them to disrupt estrogen metabolism. One of the by-products of the unfriendly “bugs” in the intestines is that the estrogen metabolites can’t be excreted and they build up in your tissues over time, causing trouble.

Terminology and classification Leukaemias are traditionally classified into four main groups: • acute lymphoblastic leukaemia (ALL) • acute myeloid leukaemia (AML) • chronic lymphocytic leukaemia (CLL) • chronic myeloid leukaemia (CML). In acute leukaemia there is proliferation of primitive stem cells leading to an accumulation of blasts, predominantly in the bone marrow, which causes bone marrow failure. In chronic leukaemia the malignant clone is able to differentiate, resulting in an accumulation of more mature cells. Lymphocytic and lymphoblastic cells are those derived from the lymphoid stem cell (B cells and T cells). Myeloid refers to the other lineages, i.e. precursors of red cells, granulocytes, monocytes and platelets (see Fig. 24.2, p. 989). The diagnosis of leukaemia is usually suspected from an abnormal blood count, often a raised white count, and is confirmed by examination of the bone marrow. This includes the morphology of the abnormal cells, analysis of cell surface markers (immunophenotyping), clone-specific chromosome abnormalities and molecular changes. These results are incorporated in the World Health Organization (WHO) classification of tumours of haematopoietic and lymphoid tissues; the subclassification of acute leukaemias is shown in Box 24.47. The features in the bone marrow not only provide an accurate diagnosis but also give valuable prognostic information, allowing therapy to be tailored to the patient’s disease.

Fibroblasts are the main cells in connective tissue and are found in all sorts of organs including skin. They’re really easy to extract and they grow very easily in culture, so are a great source of cells for experiments.

He was a little monster,” Bob said, laughing, about Steve as a child. The main difficulty wasn’t unruly behavior. It was Steve’s insatiable curiosity about the bush and the wildlife in it. “For the first few months, when he was a baby, I could put Steve down and he would stay where I put him,” Lyn told me. “But after he started to get around on his own, it was all over. I would find him either on the roof or up in some tree.” When the family headed off on a trip, usually to North Queensland on wildlife jaunts, Steve could always be counted on to be elsewhere when they were ready to go. They would find him next to the nearest stream, snagging yabbies or turning over bits of wood to see what was hidden underneath. “He was never where we wanted him to be,” Lyn recalled with a laugh. Steve’s childhood was “family, wildlife, and sport,” he told me. He played rugby league for the Caloundra Sharks in high school and was picked to play rugby for the Queensland Schoolboys and represent the state, but he chose to go on a field trip with his dad to catch reptiles instead. Sometimes sport and wildlife mixed in unexpected ways. Both was an expert badminton player, and a preteen Steve decided to layout a badminton court in the family’s backyard one day. He had a brolga as a friend, a large bird that he called Brolly. Brolly objected to Steve rearranging her territory. She waited until his back was turned and then attacked. Wham! A brolga’s beak is a fearsome weapon, and Brolly’s slammed into the back of little Stevo’s head. His bird friend knocked him out cold. “Go ahead, feel it,” Steve said after regaling me with this story. He bent his head. I could still feel a knot of scar tissue, a souvenir of the brolga attack years earlier.

An infection or inflammation of the larynx is known as laryngitis (lar-in-JI .-tis). It commonly affects the vibrational qualities of the vocal folds. Hoarseness is the most familiar result. Mild cases are temporary and seldom serious. However, bacterial or viral infections of the epiglottis can be very dangerous. The resulting swelling may close the glottis and cause suffocation. This condition, acute epiglottitis (ep-ih-glot-TI .-tis), can develop rapidly after a bacterial infection of the throat. Young children are most likely to be affected. The Trachea The trachea (TRA .-ke.-uh), or windpipe, is a tough, flexible tube with a diameter of about 2.5 cm (1 in.) and a length of about 11 cm (4.33 in.) (Figure 23–6). The trachea begins anterior to vertebra C6 in a ligamentous attachment to the cricoid cartilage. It ends in the mediastinum, at the level of vertebra T5, where it branches to form the right and left main bronchi. The epithelium of the trachea is continuous with that of the larynx. The mucosa of the trachea resembles that of the nasal cavity and nasopharynx (look back at Figure 23–2a). The submucosa (sub-mu.-KO .-suh), a thick layer of connective tissue, surrounds the mucosa. The submucosa contains tracheal glands whose mucous secretions reach the tracheal lumen through a number of short ducts. The trachea contains 15–20 tracheal cartilages that stiffen the tracheal walls and protect the airway (see Figure 23–6a). They also prevent it from collapsing or overexpanding as pressure changes in the respiratory system. Each tracheal cartilage is C-shaped. The closed portion of the C protects the anterior and lateral surfaces of the trachea. The open portion of the C faces posteriorly, toward the esophagus (see Figure 23–6b). Because these cartilages are not continuous, the posterior tracheal wall can easily distort when you swallow, allowing large masses of food to pass through the esophagus. An elastic anular ligament and the trachealis, a band of smooth muscle, connect the ends of each tracheal cartilage (see Figure 23–6b). Contraction of the trachealis reduces the diameter of the trachea. This narrowing increases the tube’s resistance to airflow. The normal diameter of the trachea changes from moment to moment, primarily under the control of the sympathetic division of the ANS. Sympathetic stimulation increases the diameter of the trachea and makes it easier to move large volumes of air along the respiratory passageways.

Into every life some rain must fall and December was one of those months when the downpour was beyond reason. Ursula, my wife of 61 years, had the same problem that I had about five years ago. Namely she had inflamed intestines brought on by a gallbladder operation which caused an intestinal blockage brought on in part by having had a total of 9 intestinal operations over the years. This time it necessitated having the numerous adhesions and quite some internal scar tissue removed. It also required the surgeon to remove a part of her small intestines (with me it was two feet) before being able to glue the two severed parts together again.

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Harmony Thai Massage Houston offer a unique combination of various massage techniques such as Swedish, Deep Tissue, Thai and so on with energy work. Our Thai massage Houston main focus is to help you bring back in balance the three energies: the mind, the body and the spirit. One of the primary goals of the Swedish massage in Houston technique is to relax the entire body. This is accomplished by rubbing the muscles with long gliding strokes in the direction of blood returning to the heart. But Swedish massage therapy goes beyond relaxation.

We must remember that there’s more than one story and plot in every novel. There are at least as many stories as there are main characters, and each of these stories has to have multiple plots to keep it going – blood and bone, nerve and tissue, forgotten longing and unknown events.

Pulsed lasers produce incredibly short bursts of electromagnetic energy. For example, a pulsed femtosecond laser produces a flash of light that lasts for femtoseconds to a picosecond (a picosecond is one trillionth of a second, a femtosecond is one thousandth of a picosecond), instantly followed by another (and so on). These lasers brought about the possibility of exciting fluorophores with two photons of only half the necessary energy, but they need to arrive almost simultaneously to generate the ejection of a photon. Infrared pulsed lasers penetrate living tissue more effectively, with the advantage that fluorescence is achieved from much deeper in the tissue than normal fluorescence, where the depth of penetration is limited by multiple light scattering events. Multiphoton microscopy (mainly two photon in practice, but also feasible as three or more photons) allows imaging from as deep as a millimetre (one thousand micrometres), an improvement of several hundred micrometres over fluorescence confocal microscopy. A second advantage of two photon excitation is that it forms as a single spot in the axial plane (z axis) without the ‘hourglass’ spread of out of focus light (the point spread function) that happens with single photon excitation. This is because the actual two photon excitation will only occur at the highest concentration of photons, which is limited to the focal plane itself. Because there is no out of focus light, there is no need for a confocal pinhole, allowing more signal to reach the detector. Combined with the increased depth of penetration, and reduced light induced damage (phototoxicity) to living tissue, two photon microscopy has added a new dimension to the imaging of living tissue in whole animals. At the surface of a living brain, remarkable images of the paths of whole neurons over several hundred micrometres can be reconstructed as a 3D z section from an image stack imaged through a thinned area of the skull in an experimental animal. Endoscopes have been developed which incorporate a miniaturized two photon microscope, allowing deep imaging of intestinal epithelium, with potential to provide new information on intestinal diseases, as most of the cellular lining throughout our gut is thin enough to be imaged in this way. So far a whole range of conditions including virtually all the cancers of the digestive tract as well as inflammatory bowel disease have been investigated, reducing the need for biopsies and providing new insights as to the nature of these conditions.

A reminder to exercise and drink plenty of water: Cellular chemicals greedily tear the molecular structure of glucose apart to extract its sugary energy. This energy extraction is so violent that atoms are literally ripped asunder in the process. As in any manufacturing process, such fierce activity generates a fair amount of toxic waste. In the case of food, this waste consists of a nasty pile of excess electrons shredded from the atoms in the glucose molecules. Left alone, these electrons slam into other molecules within the cell transforming them into one of the most toxic substances known to humankind. They are called free radicals. If not quickly corralled, they will wreck havoc on the innards...causing mutations in your very DNA. The reason you don't die of electron overdose is that the atmosphere is full of breathable oxygen. The main function of oxygen is to act like an efficient electron absorbing sponge. At the same time the blood is delivering foodstuffs to your tissues, it is also carrying those oxygen sponges. Any excess electrons are absorbed by the sponges, and after a bit of molecular alchemy, are transformed into equally hazardous but now fully transportable CO2. The blood is carried back to your lungs where the CO2 leaves the blood and you breathe it out... keeping the food you eat from killing you. This is why blood has to be everywhere inside you serving as both wait staff and hazmat team. Any tissue without blood is going to starve to death, your brain included.

Our RMR decreases by about 5% every decade after thirty, mainly because of the loss of muscle mass associated with aging. Fortunately, our lean body mass can be controlled through proper nutrition and strength training. It only takes a few months of training to recover one or two decades of decrease in our RMR. Metabolically, muscle is very expensive tissue, even when it is at rest. Another way to positively influence our RMR is to provide our body with a steady flow of nutrients. The body is extremely resourceful, and during times of starvation it adapts by slowing down its RMR. It tries to save every calorie consumed by storing some as fat. Any of the common diets that severely restrict your caloric intake neglect this principal, and that is why people on those diets almost always gain at least their original weight back. When the body receives a regular flow of calories, in the form of frequent meals, it allows the RMR to remain high, and burn those very same calories off. Frequent meals also utilize the thermal effect of food. Eating temporarily cranks up your metabolism. The more meals you eat in a day, the more consistently your metabolism is boosted. You experience an increase in your RMR for about 5 hours every time you eat. This accounts for 5 – 10% of your total calorie expenditure. Over the long haul this can make quite a difference.

P3 - ten minutes of that movie, or indeed of any movie whose message is similarly dystopian about a post-aging world (Blade Runner), you will see that they set it up by insinuating, with exactly no justification and also no attempt at discussion (which is how they get away with not justifying it), that the defeat of aging will self-evidently bring about some new problem that we will be unable to solve without doing more harm than good. The most common such problem, of course, is overpopulation - and I refer you to literally about 1000 interviews and hundreds of talks I have given on stage and camera over the past 20 years, of which several dozen are online, for why such a concern is misplaced. The reason there are 1000, of course, is that most people WANT to believe that aging is a blessing in disguise - they find it expedient to put aging out of their minds and get on with their miserably short lives, however irrational must be the rationalizations by which they achieve that. Aubrey has been asked on numerous occasions whether humans should use future tech to extend their lifespans. Aubrey opines, "I believe that humans should (and will) use (and, as a prerequisite, develop) future technologies to extend their healthspan, i.e. their healthy lifespan. But before fearing that I have lost my mind, let me stress that that is no more nor less than I have always believed. The reason people call me an “immortalist” and such like is only that I recognize, and am not scared to say, two other things: one, that extended lifespan is a totally certain side-effect of extended healthspan, and two, that the desire (and the legitimacy of the desire) to further extend healthspan will not suddenly cease once we achieve such-and-such a number of years." On what people can do to advance longevity research, my answer to this question has radically changed in the past year. For the previous 20 years, my answer would have been “make a lot of money and give it to the best research”, as it was indisputable that the most important research could go at least 2 or 3x times faster if not funding-limited. But in the past year, with the influx of at least a few $B, much of it non-profit (and much of it coming from tech types who did exactly the above), the calculus has changed: the rate-limiter now is personnel. It’s more or less the case now that money is no longer the main rate-limiter, talent is: we desperately need more young scientists to see longevity as the best career choice. As for how much current cryopreservation technology will advance in the next 10-20 years, and whether it enough for future reanimation? No question about the timeframe for a given amount of progress in any pioneering tech can be answered other than probabilistically. Or, to put it more simply, I don’t know - but I think there's a very good chance that within five years we will have cryo technology that inflicts only very little damage on biological tissue, such that yes, other advances in rejuvenation medicine that will repair the damage that caused the cryonaut to be pronounced dead in the first place will not be overwhelmed by cryopreservation damage, hence reanimation will indeed be possible. As of now, the people who have been cryopreserved(frozen) the best (i.e. w/ vitrification, starting very shortly immediately after cardiac arrest) may, just possibly, be capable of revival by rewarming and repair of damage - but only just possibly. Thus, the priority needs to be to improve the quality of cryopreservation - in terms of the reliability of getting people the best preservation that is technologically possible, which means all manner of things like getting hospitals more comfortable with cryonics practice and getting people to wear alarms that will alert people if they undergo cardiac arrest when alone, but even more importantly in terms of the tech itself, to reduce (greatly) the damage that is done to cells and tissues by the cryopreservation process.

I stood in a stupor and would have continued to stand there were it not for a breeze that parted the smoke, revealing a sailor from the Vestal. It was Joe George. He had been following orders to cut the lines that tethered his ship to the Arizona so they could head to open waters. Since there was no one on the Arizona to help on our end, he was taking a fire ax and cutting the lines on his. We called to Joe through a seam in the smoke, motioning for him to throw us a monkey’s fist, which was a lightweight heaving line knotted around a metal ball and attached to a thicker rope. It was a long shot, but our desperate idea was that if we could secure a rope between the two ships, then perhaps we could make it to the Vestal. As Joe rummaged for the ball, I looked at my arms. A sheath of skin from each had peeled off and was draping them. I tore off one length of skin and threw it on the floor of the platform. Then the other. The remaining tissue was a webwork of pink and white and red, some of it black, all of it throbbing. But that didn’t matter. My focus narrowed to Joe George and the ball in his hand. He threw it, but it fell short. He gathered up the line and lobbed it again. Short once more. Joe was perhaps the strongest man in the harbor, an All-Navy boxer whom I described earlier as an “ox.” He was the only man with a prayer of getting that line to us—if he couldn’t do it, then it was impossible. The reality started to sink in: we were going to burn alive. Joe collected the rope once more. For a third time, he tossed it with all his strength. It sailed from one wounded ship to another, across flames, smoke, and carnage. I tracked it all the way and caught it in the air, pulling the smaller line until I felt the main rope. I tied the rope to the railing, cinching it tight, and Joe secured his end. The rope stretched seventy feet to span the water below us, which was forty-five feet down, slicked with fuel that had caught fire. Our only hope was to make it to the Vestal, hand over hand across the rope. But the flesh had been burned off all of our hands, and using those raw fingers and palms to get us across the chasm that separated us would be at best excruciating, and most likely impossible.

I wonder if my feelings are stunningly obvious. Probably have been since day one, when I was assigned to his desk by central Human Resources. Before I arrived, Rufus Newton Smith, half of the brains behind Braxton Newton Smith Enterprises, churned through assistants like they were made of tissue paper. I was warned I wouldn’t last more than a week, and then I would get placed elsewhere in the company. It’s been eleven months and he’s never barked at me once.

What is diabetes? The term diabetes refers to a group of diseases that affect the way your body uses blood glucose, commonly called blood sugar. Glucose is vital to your health because it’s the main source of energy for the cells that make up your muscles and tissues. It’s your body’s main source of fuel. If you have diabetes — no matter what type — it means you have too much glucose in your blood, although the reasons why may differ. And too much glucose can lead to serious problems. To understand diabetes, it helps to understand how your body normally processes blood glucose. Processing of blood glucose Blood glucose comes from two major sources: the food you eat and your liver. During digestion, glucose is absorbed into your bloodstream. Normally, it then enters your body’s cells, aided by the action of insulin. The hormone insulin comes from your pancreas. When you eat, your pancreas secretes insulin into your bloodstream. As insulin circulates, it acts like a key, unlocking microscopic doors that allow glucose to enter your cells. In this way, insulin lowers the amount of glucose in your bloodstream and prevents it from reaching high levels. As your blood glucose level drops, so does the secretion of insulin from your pancreas. Your liver acts as a glucose storage and manufacturing center. When the level of insulin in your blood is high, such as after a meal, your liver stores extra glucose as glycogen in case your cells need it later. When your insulin levels are low, for example, when you haven’t eaten in a while, your liver releases the stored glucose into your bloodstream to keep your blood sugar level within a normal range. When you have diabetes If you have diabetes, this process doesn’t work properly. Instead of being transported into your cells, excess glucose builds up in your bloodstream, and eventually some of it is excreted in your urine. This usually occurs when your pancreas produces little or no insulin, or your cells don’t respond properly to insulin, or for both reasons. The medical term for this condition is diabetes mellitus (MEL-lih-tuhs). Mellitus is a Latin word meaning “honey sweet,” referring to the excess sugar in your blood and urine. Another form of diabetes, called diabetes insipidus (in-SIP-uh-dus), is a rare condition in which the kidneys are unable to conserve water, leading to increased urination and excessive thirst. Rather than an insulin problem, diabetes insipidus results from a different hormone disorder. In this book, the term diabetes refers only to diabetes mellitus.

The svara’s main function in Karnatik music is to give us a microcosm of the larger melody. But it cannot do this entirely on its own. It does so through a process of interaction. The svara, acts with other svaras to create smaller melodic units, which in turn define the larger melody. How does a svara, the ‘micro’, express the macro? It does so by representing an aspect of the larger melody, not by its fixity or rootedness, its immobility on a scale, but through its movable nature. Therefore, every svara can move, bounce, slide, glide, shiver or skip. How and to what extent a svara can be expressive depends on the nature of the larger musical identity it is part of and the nature of the other svaras within that macro identity. Svaras in some ways are like cells in a body. The cells (svaras) are determined by the content and function of the tissues (smaller melodic units), yet the larger human being (melody as a whole) is embedded in every cell, within the DNA.

The term fat is often used interchangeably with the term lipid, but fats are actually a particular type of lipid, called triglycerides, in which three fatty acids are bound to a compound called glycerol. Fats are important in the body. They are the main form in which the body stores energy. Stored body fat is called adipose tissue. Stored fat not only provides an energy reserve but also cushions and protects internal organs. In addition, stored fat insulates the body and helps prevent heat loss in cold weather. Although lipids and fats are necessary for life, they may be harmful if they are present in the blood at high levels. Both triglycerides and the lipid called cholesterol are known to damage blood vessels if their concentrations in the blood are too high. By damaging blood vessels, triglycerides and cholesterol also increase the risk of heart disease.

L'air de la grève refroidit quand on avance vers le large. La nuit est longue, pleine de fumée, les mouches ont déserté la plage: peut-être qu'il est trop tard et que les insectes aussi dorment quand il fait si noir. Noé marche lentement. L'eau glaciale lui monte au genoux, mais elle sait nager, même dans les vagues très froides ou quand le presbytère brûle. Elle avance à reculons – dos aux flots, à fixer le village – parce qu'il y a cette ligne, juste sous le nombril: c'est terrible quand le tissu mouille jusque-là. Il vaut mieux se jeter tout le corps à l'eau d'un coup, pour ne pas sentir la barre froide monter le long du linge. De dos, le choc est moins vif contre la peau. La nuit est grise de fumée, les nuages ressemblent à des éponges de mer qui se gonflent d'orage et de pluie. Noé recule, elle s'enfonce vers le large et soudain, des mains se referment sur ses épaules. Elle ne sursaute pas.

If we examine with our naked eye an exposed muscle compartment—in a leg of lamb, say—we notice initially that all the long thread-like cells lie parallel to one another, and that the main compartment is divided into many long and narrow subcompartments by thin septa of connective tissue which hold the muscle cells in their parallel arrangement. And if we look even more closely with the help of a microscope, we will see that within each of these long parallel cells are many extremely fine strands, also parallel in arrangement, and running the length of each cell. These fine strands within the cell are called the myofibrils, and they are the actual contracting and lengthening units of the muscle cell.